Poisons (Continued from page 19)


high and will cause reactions. Slow is better and simply raise (slowly) towards the appropriate dose for the body weight of the patient. Note that I did not say age, I said body weight. We are treating the cellular structure and not the birth certicate. I will slowly work up to a higher nal dose with a 150lb child, than I will with a 110lb adult.


I would wish you good luck, but it is my expert opinion that luck has little to do with better health. Proper and valid knowledge combined with correct and orderly application of that knowledge are the keys to attaining almost every goal in life. Improved health is no different and so I wish you a combination of the desire to improve the health of your family or your patients and the necessary effort to carry out this important desire.


About The Author David Getoff is a Board Certied Clinical Nutritionist, A Board Certied Traditional Naturopath, and an elected member of both the International College of Integrative Medicine and the American College of Nutrition.


is on the teaching faculty of the American College of Integrative Medicine and Dentistry and is the Vice President of the Price-Pottenger Nutrition Foundation. Dr Gettoff has produced over a dozen video and audio educational recordings and teaches classes, lectures, and seminars for physicians, dentists, and the public. His professional website for information only (no products are sold to the public) is www.Naturopath4you.com. 


He


Thoughts at Large (Continued from page 42)


and magnesium actually part of a bigger mineral/ 


I feel very strongly that, while discussions such as what I just presented are both clinically important and in- triguing, we must never forget that this is just one of many important physiologic and biochemical imbalances that are contributing to the chief complaints we see in chronically ill patients. One aspect of this “big picture” scenario that occurs in both acute and chronically ill patients is that the impact of stress physiology in ailing populations is not limited to potassium and magnesium imbalances. Other key electrolytes and minerals are also affected. This important point was well illustrated in the paper “Stressor states and the cation crossroads” by Weber et al (Weber KT et al. J Am Coll Nutr, Vol. 29, No. 6, pp. 563-574, December 2010): “Stressor state- induced neurohormonal activation involving calcitropic hormones, such as catecholamines, parathyroid hor- mone, vitamin D, endothelin-1, and angiotensin II, leads to homeostasis gone awry to beget dyshomeostasis at cellular and molecular levels in the heart and systemic organs. This includes a dyshomeostasis of electrolytes and cations. The cation crossroad involves monovalent and divalent cations. It can be schematically envisaged and represented by a complex set of highways, roadways, and byways as frequently invoked by stressor states.


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The acute stressor state present in critically ill patients is accompanied by dyshomeostasis of a whole host of electrolytes and trace elements manifested contemporaneously at the time of or shortly after hospital admission. Effector hormones of the HPA axis and the adrenergic nervous system and renin-angiotensin- aldosterone system collectively orchestrate the concordant appearance of hypokalemia, ionized hy-pocalcemia and hypomagnesemia, hypozincemia, and hy-poselenemia.”


A Final Thought


In this series I have been doing my best to convey to you the underappreciated issue of potassium deciency in your chronically ill patients. In part III, though, I wanted to convey that, even though the importance of potassium optimization cannot be over emphasized, we must not forget that issues of magnesium deciency and a whole host of other mineral deciencies are intricately intertwined in your chronically ill patients. In part IV, the nal installment of this series, I will explore the important but vastly under-appreciated role optimal protein and amino acid metabolism in acid/alkaline balance. 


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